STAVIS 50 is an aqueous suspension of the C17 a-alkylated steroid
stanozolol, an oral

androgen derived from dihydrotestosterone. STAVIS 50 acts on
androgen receptors to

promote anabolism through increased nitrogen retention and
protein synthesis in muscle

tissue. STAVIS 50 75 is a strong anabolic substance with androgenic
action. Stanozolol

does not convert to estrogen and therefore does not produce
typical estrogen mediated side

effects such as water retention. While chemically identical
to oral stanozolol, Stanol-AQ is

injected IM eliminating the first pass of liver metabolism
of its oral counterpart reducing

stress on the liver. Stanozolol reduces SHBG increasing free
testosterone levels.


Anabolic steroids are synthetic derivatives of the natural
steroid testosterone. Stanozolol has

been demonstrated to increase LDL and decrease HDL with
serum lipid values returning to

baseline after cessation of use. Hereditary angioedema (HAE)
is an autosomal dominant

disorder caused by a deficient or nonfunctional C1 esterase
inhibitor (C1 INH) and is

clinically characterized by episodes of swelling of the
face, extremities, genitalia, bowel wall,

and upper respiratory tract. In small clinical studies,
stanozolol was effective in controlling

the frequency and severity of attacks of angioedema and in increasing
serum levels of C1

INH and C4. Stanozolol is not effective in stopping HAE
attacks while they are underway. The

effect of stanozolol on increasing serum levels of C1 INH
and C4 may be related to an

increase in protein anabolism.


Hereditary Angioedema: for prophylactic use to decrease
frequency and severity of attacks

of angioedema.

Muscle Anabolism: for adjunctive therapy in patients for
weight gain following severe

muscular atrophy associated with extensive surgery, chronic
infections, long term

hospitalization, or severe trauma.

Corticosteroid Atrophy: to reduce muscle wasting during
prolonged corticosteroid use.


Not for use in female patients due to risk of virilization
and fetal harm.

Male patients with known or suspected carcinoma of the
breast, prostate, or testis.

Patients with hypercalcaemia as anabolic steroids may
stimulate osteolytic bone resorption.

Patients with cardiovascular disorders, renal or hepatic
impairment, epilepsy, migraines, or

diabetes mellitus.

Nephrosis or the nephrotic phase of nephritis.




Anabolic steroids may cause suppression of clotting factors
II, V, VII and X and an increase in

prothrombin time.

Anabolic steroids may increase sensitivity to
anticoagulants. Dosage of anticoagulants may

have to be decreased in order to maintain the prothrombin
time at the desired therapeutic


Oral hypoglycemic dosage may need adjustment in diabetic
patients who receive anabolic


Patients should be monitored for hepatotoxicity and


Hepatic: Cholestatic jaundice with rarely, hepatic necrosis
and death. Hepatocellular

neoplasms and peliosis hepatis have been reported in
association with long term

androgenic anabolic steroid use. Reversible changes in liver
function tests also occur

including increased bromsulphalein (BSP) retention and
increases in serum bilirubin,

glutamic oxaloacetic transaminase (SGOT), and alkaline

Genitourinary System (post pubertal men): Inhibition of
testicular functions, testicular

atrophy, and oligospermia, impotence, chronic priapism,
epididymitis and bladder irritability.

Genitourinary System (Women): Clitoral enlargement,
menstrual irregularities.

In both sexes: increased or decreased libido.

CNS: Habituation, excitation, insomnia, and depression.

Hematologic: Bleeding in patients on concomitant
anticoagulant therapy.

Hair: Hirsutism and male pattern baldness in those
genetically predisposed.

Other: Acne, oily skin, electrolytic retention, reversible
changes in serum lipids.


Serum Cholesterol, HDL, LDL, TG. Hemoglobin and Hematocrit,
Hepatic function tests –


Prostatic specific antigen – PSA, Testosterone: total, free,
and bioavailable.

Dihydrotestosterone & Estradiol

Male patients over 40 should undergo a digital rectal
examination and evaluate PSA prior to

androgen use. Periodic evaluations of the prostate should
continue while on androgen

therapy, especially in patients with difficulty in urination
or with changes in voiding habits.


Muscle anabolism: 50 – 100mg injected IM every 2 days for a
duration of 4 weeks.

Hereditary angioedema: as prescribed by physician.

The use of anabolic steroids is associated with serious
adverse reactions. Such reactions

are often dose dependent. Physicians are urged to treat
patients with the lowest possible

effective dose.


STAVIS 50 (50mg/ml) – 10 ampules of 1ml each

Each ml contains: 
Stanozolol Suspension50 mg